机构:[1]Department of Cardiology, Boston Children's Hospital, Boston, MA, USA.[2]Division of Newborn Medicine, Boston Children's Hospital, Boston, MA, USA.[3]Department of Pharmacology, Sichuan University West China School of Basic Sciences and Forensic Medicine, Chengdu, Sichuan, China.[4]Department of Anesthesiology, New York University School of Medicine, New York, NY, USA.[5]Division of Cardiovascular Medicine, Beth Israel Deaconess Medical Center, Boston, MA, USA.[6]Transgenic Technology Laboratory, Cancer Research UK Beatson Institute, Glasgow, UK.
This work was supported by funding from the U.S. Department of Defense [W81XWH2110445], National Institutes of Health [R01HL128694], the Barth Syndrome Foundation, and charitable donations from Boston Children’s Hospital Department of Cardiology.
语种:
外文
PubmedID:
中科院(CAS)分区:
出版当年[2023]版:
大类|2 区生物学
小类|3 区生化与分子生物学3 区遗传学
最新[2023]版:
大类|2 区生物学
小类|3 区生化与分子生物学3 区遗传学
第一作者:
第一作者机构:[1]Department of Cardiology, Boston Children's Hospital, Boston, MA, USA.
通讯作者:
通讯机构:[1]Department of Cardiology, Boston Children's Hospital, Boston, MA, USA.[6]Transgenic Technology Laboratory, Cancer Research UK Beatson Institute, Glasgow, UK.
推荐引用方式(GB/T 7714):
Wang Suya,Yazawa Erika,Keating Erin,et al.Genetic modifiers modulate phenotypic expression of tafazzin deficiency in a mouse model of Barth syndrome[J].Human molecular genetics.2023,doi:10.1093/hmg/ddad041.
APA:
Wang Suya,Yazawa Erika,Keating Erin,Mazumdar Neil,Hauschild Alexander...&Pu William T.(2023).Genetic modifiers modulate phenotypic expression of tafazzin deficiency in a mouse model of Barth syndrome.Human molecular genetics,,
MLA:
Wang Suya,et al."Genetic modifiers modulate phenotypic expression of tafazzin deficiency in a mouse model of Barth syndrome".Human molecular genetics .(2023)